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Fig. 1 | Stem Cell Research & Therapy

Fig. 1

From: Competing endogenous RNA network analysis of Turner syndrome patient-specific iPSC-derived cardiomyocytes reveals dysregulation of autosomal heart development genes by altered dosages of X-inactivation escaping non-coding RNAs

Fig. 1

Characterization of wild-type (WT) and Turner syndrome (TS)-specific induced pluripotent stem cell (iPSC) lines. A Representative colony morphology of WT- and TS-iPSCs. B Both WT- and TS-iPSC colonies were positive for alkaline phosphatase staining. C Karyotype analysis showing X chromosome number of WT- and TS-iPSCs. D Immunofluorescence of the pluripotency markers OCT4, SOX2, SSEA4 and TRA-60 in WT- and TS-iPSCs. E Embryoid body (EB) differentiation of WT- and TS-iPSCs in vitro. F Immunofluorescence of germ layer markers nestin, SMA and AFP in WT- and TS-iPSCs. G Teratoma formation assay showing both WT- and TS-iPSCs differentiates into tissues of the three germ layers in vivo. Scale bar = 100 μm

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Stem Cell Research & Therapy

ISSN: 1757-6512