Fig. 3From: Human decidua basalis mesenchymal stem/stromal cells protect endothelial cell functions from oxidative stress induced by hydrogen peroxide and monocytesProliferation of HUVEC measured by MTS. As compared to untreated HUVEC, the proliferation of HUVEC significantly increased in response to H2O2 alone or with different concentrations (1%, 5%, and 25%) of CMDBMSC in the presence of 100 μM H2O2 (a) and with different ratios of DBMSC to HUVEC (1:1, 1:5, and 1:10) in the presence of 100 μM H2O2 (b). As compared to HUVEC treated with H2O2 (HUVEC + H2O2), the proliferation of HUVEC significantly increased in response to different concentrations (1%, 5% and 25%) of CMDBMSC in presence of 100 μM H2O2 (a) and with different ratios of DBMSC to HUVEC (1:1, 1:5, and 1:10) in presence of 100 μM H2O2 (b). As compared to untreated HUVEC, HUVEC proliferation significantly increased in response to different concentrations (1%, 5%, and 25%) of CMDBMSC (a), and at a high ratio of DBMSC to HUVEC (1:1) (b). As compared to HUVEC cultured with CMDBMSC, HUVEC proliferation did not significantly change in response to H2O2 and CMDBMSCs, P > 0.05 (a). As compared to HUVEC cultured with low ratios of DBMSCs to HUVEC (1:5 and 1:10), HUVEC proliferation significantly increased in response to H2O2 and DBMSCs (b). *P < 0.05. Bars represent standard errors. Each experiment was performed in triplicate and repeated for five times with five independent preparations of DBMSCs and HUVECBack to article page