Fig. 8From: S100B is required for maintaining an intermediate state with double-positive Sca-1+ progenitor and vascular smooth muscle cells during neointimal formationWorking model of S100B the process of neointimal formation. S100B triggered neointimal formation in the injured arteries by maintaining the intermediate state of double-positive Sca-1+ progenitor cells and VSMCs that was associated with RAGE and/or SDF-1α/CXCR4Back to article page