Fig. 7
![Fig. 7](http://media.springernature.com/full/springer-static/image/art%3A10.1186%2Fs13287-023-03562-7/MediaObjects/13287_2023_3562_Fig7_HTML.png)
Depletion of CB11b in SVF modifies intestinal immune cells population. A neutrophil (CD11b+Ly6G+), cDC1 (CD11b−CD11+CD103+) and cDC2 (CD11b+CD11+CD103+), CD4+ and CD8+ population. B Percentage of monocytes phenotype CD11b+Ly6C+CCR2+, CD11b+Ly6C+MCHII− CX3Cr1− (P1), CD11b+Ly6C−MCHII+CX3Cr1int (P2), CD11b+Ly6C−MCHII+CX3Cr1+ (P3) and CD11b+Ly6C−MCHII+ F4/80+ are analysis among CD45+ cells from ileal lamina propria of control and at day 7 after irradiation with or without SVF treatment depleted or not of CD11b. The data are represented by mean ± SEM (for each groups n = 6). C Real-time PCR analysis of IL-1β, IL-6 and IL-1Rn of ileal tissue. The comparative threshold cycle (Ct) method was used, and the delta Ct comparison was used to compare gene expression in cells. P values were calculated by ANOVA with Tukey correction; *p < 0.05; **p < 0.01; ***p < 0.001 compared with the WT control mice; #p < 0.05; ##p < 0.01; ###p < 0.001 compared with the WT irradiated mice; $$$p < 0.001 compared with the WT irradiated treated SVF mice