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Correction to: PARP1 inhibitor (PJ34) improves the function of aging-induced endothelial progenitor cells by preserving intracellular NAD+ levels and increasing SIRT1 activity

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The original article was published in Stem Cell Research & Therapy 2018 9:224

Correction

The original article [1] contains an error regarding the erroneous inclusion of 3 μl as a parameter in the x-axis of Fig. 2c; the correct version of Fig. 2c can instead be seen below.

Fig. 2
figure1

Effects of H2O2 and PJ34 treatment on EPC protein expression. a, b Expression of sirtuin 1 (SIRT1), poly (ADP-ribose) polymerase 1 (PARP1), and poly ADP-ribose (PAR) as analyzed by Western blot. c, d Expression of PARP1, PAR, and acetylated (ac)-p53 as analyzed by Western blot. *P < 0.05, **P < 0.01, ***P < 0.001, versus the control

Reference

  1. 1.

    Zha S, et al. PARP1 inhibitor (PJ34) improves the function of aging-induced endothelial progenitor cells by preserving intracellular NAD+ levels and increasing SIRT1 activity. Stem Cell Res Ther. 2018;9:224 https://doi.org/10.1186/s13287-018-0961-7.

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Correspondence to Fang Liu.

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Zha, S., Li, Z., Cao, Q. et al. Correction to: PARP1 inhibitor (PJ34) improves the function of aging-induced endothelial progenitor cells by preserving intracellular NAD+ levels and increasing SIRT1 activity. Stem Cell Res Ther 9, 269 (2018) doi:10.1186/s13287-018-1019-6

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