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Correction to: PARP1 inhibitor (PJ34) improves the function of aging-induced endothelial progenitor cells by preserving intracellular NAD+ levels and increasing SIRT1 activity
Stem Cell Research & Therapy volume 9, Article number: 269 (2018)
Correction
The original article [1] contains an error regarding the erroneous inclusion of 3 μl as a parameter in the x-axis of Fig. 2c; the correct version of Fig. 2c can instead be seen below.
Effects of H2O2 and PJ34 treatment on EPC protein expression. a, b Expression of sirtuin 1 (SIRT1), poly (ADP-ribose) polymerase 1 (PARP1), and poly ADP-ribose (PAR) as analyzed by Western blot. c, d Expression of PARP1, PAR, and acetylated (ac)-p53 as analyzed by Western blot. *P < 0.05, **P < 0.01, ***P < 0.001, versus the control
Reference
Zha S, et al. PARP1 inhibitor (PJ34) improves the function of aging-induced endothelial progenitor cells by preserving intracellular NAD+ levels and increasing SIRT1 activity. Stem Cell Res Ther. 2018;9:224 https://doi.org/10.1186/s13287-018-0961-7.
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Zha, S., Li, Z., Cao, Q. et al. Correction to: PARP1 inhibitor (PJ34) improves the function of aging-induced endothelial progenitor cells by preserving intracellular NAD+ levels and increasing SIRT1 activity. Stem Cell Res Ther 9, 269 (2018). https://doi.org/10.1186/s13287-018-1019-6
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DOI: https://doi.org/10.1186/s13287-018-1019-6